De Marco, F., Bucaj, E., Foppoli, C., Fiorini, A., Blarzino, C., Filipi, K., Giorgi, A., Schinina, M.E., Di Domenico, F., Coccia, R., Butterfield, D.A. and Perluigi, M. (2012) Oxidative Stress in HPV-Driven Viral Carcinogenesis: Redox Proteomics Analysis of HPV-16 Dysplastic and Neoplastic Tissues. PLoS ONE, 7, e34366.
http://dx.doi.org/10.1371/journal.pone.0034366

has been cited by the following article:

• TITLE: Prevention of Iatrogenic Cervical Cancer

  AUTHORS: Rudolf Klimek

  KEYWORDS: Cervical Cancer, Cancer Cell, HPV

  JOURNAL NAME: Open Journal of Obstetrics and Gynecology, Vol.5 No.14, November 30, 2015

  ABSTRACT: Cervical carcinogenesis consists of natural occurring spontaneous cellular processes which may lead to self-organized dissipative structures of cervical cancers what was first explained in 1977 after several years of my biochemical, biophysical, hormonal and clinical studies. That was possible thanks to monograph “Biochemie der Tumoren” written in 1942 by Nobel Prize winner H. von Euler with my master Prof. B. Skarzynski. Today I express my gratitude to Nobelist Harald zur Hausen and his team for they discovered the nuclide sequences of HPV in genomes of cervical cancer cells which opened the possibility to describe the causal role of information in formula of reality. Vaccines built from the protein capsid of HPV have proved only the pathogenic information about the virus because of its lack of DNAs. All the theories of carcinogenesis have properly described this event from methodologically different point of view. The point is that one should understand the thermodynamic rules underlying each of these approaches. Neoplasms are self-organized from the cells of the patient, who did not provide the necessary conditions for cellular metabolism as defined in the moment of appearance of its zygote. In light of medical thermodynamics all oncogenic factors can divide into sufficient or necessary to events for creating a dissipathogenic cellular status. Cervical cancer is a tumor associated with the human papillomavirus as only its pathogenic dissipathogenic factors, but the genome of cervical carcinoma cells maybe the original source of many types of HPV from the peeled off cancer cells of the uterine cervix. Many things are known to increase the risk of carcinogenesis which as a natural process is an alternative of cellular or social death. Neoplasm cell is an effect of carcinogenesis, but not a causal point at which it begins its existence.