Jahanyar, J., Youker, K.A., Loebe, M., Assad-Kottner, C., Koerner, M.M., Torre-Amione, G. and Noon, G.P. (2007) Mast cell-derived cathepsin G: A possible role in the adverse remodeling of the failing human heart. Journal of Surgical Research, 140, 199-203. doi:10.1016/j.jss.2007.02.040

has been cited by the following article:

• TITLE: The activities of endothelial elastase and cathepsin G in rats at oxidative stress caused by heavy metals salts

  AUTHORS: Nickolaj F. Starodub, Ljubov M. Samokhina

  KEYWORDS: Endothelial Elastase; Cathepsin G; Cobalt Chloride; Mercury Chloride; Rats

  JOURNAL NAME: Advances in Biological Chemistry, Vol.3 No.2, April 30, 2013

  ABSTRACT: CoCl2 introduction increased cathepsin G activity in the heart and liver as well as endothelial elastase (EEl) in kidney that indicated the development of destructive processes. CoCl2 introduction decreased EEl and cathepsin G activities in blood serum and cathepsin G in lungs. HgCl2 injection decreased EEl in blood serum, heart, liver, kidney and cathepsin G in blood serum. These decreasing of proteinases activities may be caused by cytotoxic effects of heavy metals and/or the inclusion of these proteases in the destructive processes and absence of their synthesis and/or release.