Mitsuhashi, A., Goto, H., Kuramoto, T., Tabata, S., Yukishige, S., Abe, S., Hanibuchi, M., Kakiuchi, S., Saijo, A., Aono, Y., Uehara, H., Yano, S., Ledford, J.G., Sone, S. and Nishioka, Y. (2013) Surfactant Protein A Suppresses Lung Cancer Progression by Regulating the Polarization of Tumor-Associated Macrophages. The American Journal of Pathology, 182, 1843-1853.
http://dx.doi.org/10.1016/j.ajpath.2013.01.030

has been cited by the following article:

• TITLE: Morphofunctional Characteristics of Pulmonary Surfactant System and Its Effect on Immune Cells in Influenza A (H1N1) Pathogenesis

  AUTHORS: A. V. Kovner, O. V. Potapova, V. A. Shkurupy, A. M. Shestopalov

  KEYWORDS: Influenza A (H1N1) Virus, Pulmonary Surfactant system, SP-A, M1/M2 Macrophages, Antiviral Defense

  JOURNAL NAME: Open Journal of Pathology, Vol.6 No.1, January 6, 2016

  ABSTRACT: There is an annual increase of influenza-related SARI cases in winter months. Despite the high relevance of this problem, influenza pathogenesis and the role of surfactant system and its SP-A (surfactant protein A) enzyme in antiviral defense remain poorly understood. SP-A activates macrophage M1 polarization and triggers an antiviral response due to the activation of T-cells and dendritic cells. Therefore, surfactant system is an important element of infection protection and a promising therapeutic target.