Gupta, N.K., de Lemos, J.A., Ayers, C.R., Abdullah, S.M., McGuire, D.K. and Khera, A. (2012) The Relationship between C-Reactive Protein and Atherosclerosis Differs on the Basis of Body Mass Index: The Dallas Heart Study. Journal of the American College of Cardiology, 60, 1148-1155.
http://dx.doi.org/10.1016/j.jacc.2012.04.050

has been cited by the following article:

• TITLE: Pro-Inflammatory Substances and Cognition in the Dallas Heart Study

  AUTHORS: Keith A. Bernardo, Heidi C. Rossetti, Myron F. Weiner, Colin Munro Cullum, James De Lemos, Laura H. Lacritz

  KEYWORDS: Montreal Cognitive Assessment, Cognition, Inflammation, Dallas Heart Study

  JOURNAL NAME: Open Journal of Psychiatry, Vol.6 No.1, January 19, 2016

  ABSTRACT: Cognitive decline in late adulthood might be partially mediated by subclinical generalized vascular disease. If so, atherogenic factors such as pro-inflammatory cytokines might be mid-life targets for prevention or treatment. Dallas Heart Study subjects (n = 997; mean age = 42.94 ± 10.2 yrs) underwent blood assays of pro-inflammatory biomarkers associated with atherosclerosis and 8 years later completed a cognitive outcome measure, the Montreal Cognitive Assessment (MoCA). Markers included C-reactive protein (CRP), Interleukin-18 (IL-18), Lipoprotein-associated phospholipase (LP-PLA2), and Monocyte Chemoattractant Protein (MCP-1), with Apolipoprotein E4 (ApoE4) as a potential modifier. We found weak evidence for LP-PLA2 and CRP as predictors of cognitive scores. No relationship was found between elevated MCP-1, IL-18 and cognition. Presence of the ApoE4 allele did not impact the relationship between biomarkers and cognitive function. Levels of atherogenesis-related pro-inflammatory blood biomarkers did not predict cognitive function in middle-aged adults after an interval of 8 years.