Martin, L., Latypova, X. and Terro, F. (2011) Post-Translational Modifications of Tau Protein: Implications for Alzheimer’s Disease. Neurochemistry International, 58, 458-471.
http://dx.doi.org/10.1016/j.neuint.2010.12.023

has been cited by the following article:

• TITLE: Strategizing the Development of Alzheimer’s Therapeutics

  AUTHORS: Justin Davis, Robin Couch

  KEYWORDS: Drug Discovery, Alzheimer’s Disease, Target Identification, Therapeutics, Review

  JOURNAL NAME: Advances in Alzheimer's Disease, Vol.3 No.3, September 12, 2014

  ABSTRACT: Alzheimer’s Disease is a complex, progressive condition with symptoms that do not reveal themselves until significant changes to neuronal morphology have already occurred. The delayed manifestation of cognitive decline makes determination of the true etiological origins difficult. As a result, identification of ideal drug targets becomes seemingly impossible. The existing treatments for Alzheimer’s Disease may temporarily suppress the rate of cognitive decline, but do little to slow or halt neuronal decay. While many believe that the current approaches to identifying a cure for the disease are too narrow-minded, focusing heavily on the physical manifestations of the diseased brain such as amyloid plaques and neurofibrillary tangles, this review asserts the status of Alzheimer’s research as rational and multi-faceted.