Kido, J., Kido, R., Suryono, Kataoka, M., Fagerhol, M.K. and Nagata, T. (2003) Calprotectin Release from Human Neutrophils Is Induced by Porphyromonas gingivalis Lipopolysaccharide via the CD-14-Toll-Like Receptor-Nuclear Factor KappaB Pathway. Journal of Periodontal Research, 38, 557-563.

has been cited by the following article:

• TITLE: Oral Bacteria Are Responsible for the Etiology of Rheumatoid Arthritis

  AUTHORS: Mesut Ogrendik

  KEYWORDS: Rheumatoid Arthritis, Etiology, Oral Bacteria

  JOURNAL NAME: Open Journal of Rheumatology and Autoimmune Diseases, Vol.4 No.3, August 8, 2014

  ABSTRACT: A significant association has been shown to exist between periodontitis and rheumatoid arthritis (RA) in observational studies. Some intervention studies have suggested that periodontal treatment can reduce serum inflammatory biomarkers such as C-reactive protein, or erythrocyte sedimentation rate. Serum antibodies to oral bacteria have been found at increased levels in RA patients. In addition, DNAs from these bacteria have been identified from samples of RA patients. Several antibiotics are effective on RA. Finally, oral bacteria are responsible for the etiology of RA.