"Normal Human Cell Conversion to 3-D Cancer-like Growth: Genome Damage, Endopolyploidy, Senecence Escape, and Cell Polarity Change/Loss"
written by Kirsten H. Walen,
published by Journal of Cancer Therapy, Vol.2 No.2, 2011
has been cited by the following article(s):
  • Google Scholar
  • CrossRef
[1] Selective removal of age-modified cells for treatment of atherosclerosis
2019
[2] Product and method for treating sarcopenia
2019
[3] Genomic Instability in Cancer II: 4N-Skewed (90°) Reductive Division via Fragile Sites to Fitness Increase for Solid and Hematological Cancer Beginnings
2019
[4] Genomic Instability in Cancer I: DNA-Repair Triggering Primitive Hereditary 4n-Skewed, Amitotic Division-System, the Culprit in EMT/MET/Metaplasia Cancer …
2018
[5] Cancer cells arise from bacteria
2018
[6] Mitotic Slippage Process Concealed Cancer-Sought Chromosome Instability Mechanism (S-CIN)
2017
[7] Cancer Prevention? Fundamental Genomic Alterations Are Present in Preneoplasia, Including Function of High Frequency Selected Mutations (HFSMs)
2016
[8] Wound Healing Is a First Response in a Cancerous Pathway: Hyperplasia Developments to 4n Cell Cycling in Dysplasia Linked to Rb-Inactivation
Journal of Cancer Therapy, 2015
[9] Neoplastic-Like Cell Changes of Normal Fibroblast Cells Associated with Evolutionary Conserved Maternal and Paternal Genomic Autonomous Behavior …
2014
[10] Haploidization of Human Diploid Metaphase Cells: Is This Genome Reductive Mechanism Opperational in Near-Haploid Leukemia
2014
[11] Neoplastic-Like CELL Changes of Normal Fibroblast Cells Associated with Evolutionary Conserved Maternal and Paternal Genomic Autonomous Behavior (Gonomery)
Journal of Cancer Therapy, 2014
[12] Normal Human Cells Acquiring Proliferative Advantage to Hyperplasia-Like Growth-Morphology: Aberrant Progeny Cells Associated with Endopolyploid and Haploid …
Cancer and Clinical Oncology, 2013
[13] Senescence Arrest of Endopolyploid Cells Renders Senescence into One Mechanism for Positive Tumorigenesis
Tumor Dormancy, Quiescence, and Senescence, Volume 1. Springer Netherlands,?, 2013
[14] into One Mechanism for Positive Tumorigenesis
Tumor Dormancy, Quiescence, and Senescence, Volume 1: Aging, Cancer, and Noncancer Pathologies?, 2013
[15] Normal Human Cells Acquiring Proliferative Advantage to Hyperplasia-Like Growth-Morphology: Aberrant Progeny Cells Associated with Endopolyploid and Haploid Divisions
Cancer and Clinical Oncology, 2013
[16] Haploidization of Human Diploid Metaphase Cells: Is This Genome Reductive Mechanism Opperational in Near-Haploid Leukemia?
Journal of Cancer Therapy, 2013
[17] Normal Human Cells Acquiring Proliferative Advantage to Hyperplasia-Like Growth-Morphology: Aberrant Progeny Cells Associated with Endopolyploid …
2013
[18] Genome reversion process of endopolyploidy confers chromosome instability on the descendent diploid cells
Cell biology international?, 2012