Absence of Nitric Oxide Synthase 3 Increases Amyloid <i>β</i>-Protein Pathology in Tg-5xFAD Mice

ISSN Print: 2158-2912
ISSN Online: 2158-2947
www.scirp.org/journal/nm
E-mail: nm@scirp.org

"Absence of Nitric Oxide Synthase 3 Increases Amyloid β-Protein Pathology in Tg-5xFAD Mice"
written by Zishuo Ian Hu, Ann Marie E. Kotarba, William E. Van Nostrand,
published by Neuroscience and Medicine, Vol.4 No.2, 2013

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[2]	Proinflammatory cytokines induce accumulation of glypican-1-derived heparan sulfate and the C-terminal fragment of β-cleaved APP in autophagosomes of dividing neuronal cells Glycobiology, 2020 DOI:10.1093/glycob/cwaa011

[3]	The cyanobacterial neurotoxin β-N-methylamino-l-alanine prevents addition of heparan sulfate to glypican-1 and increases processing of amyloid precursor protein in dividing neuronal cells Experimental Cell Research, 2019 DOI:10.1016/j.yexcr.2019.03.041

[4]	Mouse models of Alzheimer’s disease cause rarefaction of pial collaterals and increased severity of ischemic stroke Angiogenesis, 2018 DOI:10.1007/s10456-018-9655-0

[5]	A protocol for quantitative analysis of murine and human amyloid-β 1-40 and 1-42 Journal of Neuroscience Methods, 2017 DOI:10.1016/j.jneumeth.2017.07.022

[6]	Hypoxia induces NO-dependent release of heparan sulfate in fibroblasts from the Alzheimer mouse Tg2576 by activation of nitrite reduction Glycobiology, 2016 DOI:10.1093/glycob/cww007