Curcumin Protects SK-N-MC Cells from H<sub>2</sub>O<sub>2</sub>-Induced Cell Death by Modulation of Notch Signaling Pathway

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ISSN Print: 2325-7776
ISSN Online: 2325-7792
www.scirp.org/journal/cellbio
E-mail: cellbio@scirp.org

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"Curcumin Protects SK-N-MC Cells from H₂O₂-Induced Cell Death by Modulation of Notch Signaling Pathway"
written by Maryam Kamarehei, Razieh Yazdanparast, Safie Aghazadeh,
published by CellBio, Vol.3 No.2, 2014

has been cited by the following article(s):

[1]	Molecular mechanisms of developmental pathways in neurological disorders: a pharmacological and therapeutic review
	Open …, 2022

[2]	Toxicity and Selective Biochemical Assessment of Quercetin, Gallic Acid, and Curcumin in Zebrafish
	2019

[3]	Radix Ophiopogonis polysaccharide extracts alleviate MPP+-induced PC-12 cell injury through inhibition of Notch signaling pathway
	2018

[4]	Wound healing property of isolated compounds from Boesenbergia kingii rhizomes
	Journal of ethnopharmacology, 2016

[1]	The Emerging Role of Curcumin in Inducing Neuronal Trans-differentiation of Mesenchymal Stem Cells: a Review Regenerative Engineering and Translational Medicine, 2023 DOI:10.1007/s40883-023-00300-6

[2]	The Emerging Role of Curcumin in Inducing Neuronal Trans-differentiation of Mesenchymal Stem Cells: a Review Regenerative Engineering and Translational Medicine, 2023 DOI:10.1007/s40883-023-00300-6

[3]	Molecular mechanisms of developmental pathways in neurological disorders: a pharmacological and therapeutic review Open Biology, 2022 DOI:10.1098/rsob.210289

[4]	Molecular mechanisms of developmental pathways in neurological disorders: a pharmacological and therapeutic review Open Biology, 2022 DOI:10.1098/rsob.210289

[5]	Molecular mechanisms of developmental pathways in neurological disorders: a pharmacological and therapeutic review Open Biology, 2022 DOI:10.1098/rsob.210289

[6]	Toxicity and Selective Biochemical Assessment of Quercetin, Gallic Acid, and Curcumin in Zebrafish Biological and Pharmaceutical Bulletin, 2019 DOI:10.1248/bpb.b19-00296

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