"Cks1: Structure, Emerging Roles and Implications in Multiple Cancers"
written by Vinayak Khattar, Jaideep V. Thottassery,
published by Journal of Cancer Therapy, Vol.4 No.8, 2013
has been cited by the following article(s):
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[1] Cks1 regulates human hepatocellular carcinoma cell progression through osteopontin expression
2019
[2] FOXM1 and CKS1-novel cellular targets of the HPV oncogenes
2019
[3] MicroRNA-940 inhibits glioma cells proliferation and cell cycle progression by targeting CKS1
2019
[4] Análise do gene GBA1 em pacientes com mieloma múltiplo
2018
[5] The Cks1/Cks2 axis fine-tunes Mll1 expression and is crucial for MLL-rearranged leukaemia cell viability
Biochimica et Biophysica Acta (BBA) - Molecular Cell Research, 2018
[6] CKS1BP7, a Pseudogene of CKS1B, is Co-Amplified with IGF1R in Breast Cancers
Pathology & Oncology Research, 2017
[7] High-expressed CKS2 is associated with hepatocellular carcinoma cell proliferation though down-regulating PTEN
Pathology - Research and Practice, 2017
[8] Long noncoding RNA MALAT 1 affects the efficacy of radiotherapy for esophageal squamous cell carcinoma by regulating Cks1 expression
Journal of perinatal medicine, 2017
[9] Increased expression of Cks1 protein is associated with lymph node metastasis and poor prognosis in nasopharyngeal carcinoma
2017
[10] 3-O-(Z)-coumaroyloleanolic acid Overcomes Cks1b-induced Chemoresistance in Lung Cancer by Inhibiting Hsp90 and MEK Pathways
Biochemical Pharmacology, 2017
[11] The cyclin dependent kinase subunit Cks1 is required for infection‐associated development of the rice blast fungus Magnaporthe oryzae
Environmental microbiology, 2017
[12] Gene signature associated with benign neurofibroma transformation to malignant peripheral nerve sheath tumors
PLOS ONE, 2017
[13] CKS1BP7, a Pseudogene of CKS1B, is Co-Amplified with IGF1R in Breast Cancers.
Pathology & Oncology Research, 2017
[14] TORC1 coordinates the conversion of Sic1 from a target to an inhibitor of cyclin-CDK-Cks1
Cell discovery, 2017
[15] Hormonal and growth regulation of epithelial and stromal cells from the normal and malignant endometrium by Pigment epithelial-derived factor (PEDF)
Endocrinology, 2017
[16] Hormonal and Growth Regulation of Epithelial and Stromal Cells From the Normal and Malignant Endometrium by Pigment Epithelium-Derived Factor
Endocrinology, 2017
[17] TGF-β activates APC through Cdh1 binding for Cks1 and Skp2 proteasomal destruction stabilizing p27kip1 for normal endometrial growth
International Journal of Phytoremediation, 2016
[18] JBC Papers in Press. Published on January 11, 2016 as Manuscript M115. 711648
2016
[19] Regulation of Transient Site-specific Copy Gain by MicroRNA
2016
[20] Long noncoding RNA MALAT1 affects the efficacy of radiotherapy for esophageal squamous cell carcinoma by regulating Cks1 expression
Journal of perinatal medicine, 2016
[21] Effect of cyclin dependent kinase submit 1 on proliferation of hepatocellular carcinoma (HCC) BEL-7405 cells
2016
[22] 细胞周期激酶亚单位 1 对肝细胞癌细胞增殖能力的影响
2016
[23] 舌鳞状细胞癌组织中 Cks1, Skp2 和 p27~(kip1) 蛋白的表达及其临床意义
2015
[24] 长链非编码 RNA: 非小细胞肺癌研究的一个新领域
临床肿瘤学杂志, 2015
[25] 舌鳞状细胞癌组织中 Cks1, Skp2 和 p27 kip1 蛋白的表达及其临床意义
2015
[26] Cks1 proteasomal turnover is a predominant mode of regulation in breast cancer cells: Role of key tyrosines and lysines
International journal of oncology, 2015
[27] Hypoxia drives transient site-specific copy gain and drug-resistant gene expression
Genes & development, 2015
[28] Asparaginase treatment side-effects may be due to genes with homopolymeric Asn codons (Review-Hypothesis)
International journal of molecular medicine, 2015
[29] Análisis de la inhibición del oncogén FOXM1 en células de glioma
2015
[30] Implicaciones del estudio de inestabilidad del ciclo celular en la biología del cáncer.
Revista CENIC. Ciencias Biológicas, 2014
[31] Cks1 proteasomal degradation is induced by inhibiting Hsp90-mediated chaperoning in cancer cells
Cancer chemotherapy and pharmacology, 2014
[32] The basal transcription machinery as a target for cancer therapy
Cancer cell international, 2014