Advances in Pancreatic Cancer
Pancreatic cancer arises when cells in the pancreas, a glandular organ behind the stomach, begin to multiply out of control and form a mass. These cancerous cells have the ability to invade other parts of the body. A number of types of pancreatic cancer are known. In the present book, sixteen typical literatures about Pancreatic Cancer published on international authoritative journals were selected to introduce the worldwide newest progress, which contains reviews or original researches on Pancreatic Cancer. We hope this book can demonstrate advances in Pancreatic Cancer as well as give references to the researchers, students and other related people.
Sample Chapter(s)
Preface (148 KB)
Components of the Book:
  • Chapter 1
    Impact of multi-center diagnostic workup in patients with pancreatic cancer on repeated diagnostic investigations, time-to-diagnosis and time-to-treatment: A nationwide analysis
  • Chapter 2
    Neoadjuvant therapy or upfront surgery for resectable and borderline resectable pancreatic cancer: A meta-analysis of randomized controlled trials
  • Chapter 3
    Trends in pancreatic cancer incidence, characteristics, and outcomes in Denmark 1980–2019: A nationwide cohort study
  • Chapter 4
    Are older patients less likely to be treated for pancreatic cancer? A systematic review and meta-analysis
  • Chapter 5
    Butyrate, a postbiotic of intestinal bacteria, affects pancreatic cancer and gemcitabine response in in vitro and in vivo models
  • Chapter 6
    Discovery of a novel NUAK1 inhibitor against pancreatic cancer
  • Chapter 7
    Hereditary pancreatic cancer
  • Chapter 8
    Oncolytic viruses and pancreatic cancer
  • Chapter 9
    Pseudostellaria heterophylla Extract Polysaccharide H-1-2 Suppresses Pancreatic Cancer by Inhibiting Hypoxia-Induced AG2
  • Chapter 10
    HNF4A and GATA6 Loss Reveals Therapeutically Actionable Subtypes in Pancreatic Cancer
  • Chapter 11
    Prediction and identification of synergistic compound combinations against pancreatic cancer cells
  • Chapter 12
    Marker-free lineage tracing reveals an environment-instructed clonogenic hierarchy in pancreatic cancer
  • Chapter 13
    L3MBTL2-mediated CGA transcriptional suppression promotes pancreatic cancer progression through modulating autophagy
  • Chapter 14
    Antitumor effects of iPSC-based cancer vaccine in pancreatic cancer
  • Chapter 15
    Pancreatic cancer chemo-resistance is driven by tumor phenotype rather than tumor genotype
  • Chapter 16
    Stabilized epithelial phenotype of cancer cells in primary tumors leads to increased colonization of liver metastasis in pancreatic cancer
Readership: Students, academics, teachers and other people attending or interested in Pancreatic Cancer.
Jana S.Hopstaken
Department of Surgery, Radboud University Medical Center, Nijmegen, the Netherlands

Marc G.Besselink
Department of Surgery, Amsterdam UMC, University of Amsterdam, Cancer Center Amsterdam, Amsterdam, the Netherlands

Frank Viborg Mortensen
Department of Clinical Medicine, Aarhus University, Denmark

Mette Fugleberg Nielsen
Department of Surgery, G?dstrup Regional Hospital, Denmark

Siang-Boon Koh
Cancer Research UK Cambridge Institute, University of Cambridge, Cambridge CB2 0RE, UK

and more...
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