[1]
|
Khakh, B.S. and Burnstock, G. (2009) The double life of ATP. Scientific American, 301, 84-90.
doi:10.1038/scientificamerican1209-84
|
[2]
|
Dzeja, P. and Terzic, A. (2009) Adenylate kinase and AMP signaling networks: Metabolic monitoring, signal communication and body energy sensing. International Journal of Molecular Sciences, 10, 1729-1772.
doi:10.3390/ijms10041729
|
[3]
|
Matsuura, S., Igarashi, M., Tanizawa, Y., Yamada, M., Kishi, F., Kajii, T., Fujii, H., Miwa, S., Sakurai, M. and Nakazawa, A. (1989) Human adenylate kinase deficiency associated with hemolytic anemia. A single base substitution affecting solubility and catalytic activity of the cytosolic adenylate kinase. Journal of Biological Chemistry, 264, 10148-10155.
|
[4]
|
Gloria-Bottini, F., Antonacci, E., Cozzoli, E., De Acetis, C. and Bottini, E. (2010) The effect of genetic variability on the correlation between blood glucose and glycated hemoglobin levels. Metabolism, 60, 250-255.
doi:10.1016/j.metabol.2010.01.003
|
[5]
|
Weitkamp, L.R., Sing, C.F., Shreffler, D.C. and Guttormsen, S.A. (1969) The genetic linkage relations of adenylate kinase: Further data on the ABO-AK linkage group. American Journal of Human Genetics, 21, 600- 605.
|
[6]
|
Bottini, N., Gloria-Bottini, F., Borgiani, P., Antonacci, E., Lucarelli, P. and Bottini, E. (2004) Type 2 diabetes and the genetics of signal transduction: a study of interaction between adenosine deaminase and acid phosphatase locus 1 polymorphisms. Metabolism, 53, 995-1001.
doi:10.1016/j.metabol.2004.03.006
|
[7]
|
Fildes, R.A. and Harris, H. (1966) Genetically determined variation of adenylate kinase in man. Nature, 209, 261-263. doi:10.1038/209261a0
|
[8]
|
Spencer, N., Hopkinson, D.A. and Harris, H. (1964) Quantitative differences and gene dosage in the human red cell acid phosphatase polymorphism. Nature, 201, 299- 300. doi:10.1038/201299a0
|
[9]
|
SPSS/PC+ Version 5.0 (1992) Chicago: SPSS Inc.
|
[10]
|
Sokal, R.R. and Rohlf, J.F. (1981) Biometry, WH Freeman, New York.
|
[11]
|
Petit, P., Lajoix, A.D. and Gross, R. (2009) P2 purinergic signalling in the pancreatic beta-cell: Control of insulin secretion and pharmacology. European Journal of Pharmaceutical Sciences, 37, 67-75.
doi:10.1016/j.ejps.2009.01.007
|
[12]
|
Solini, A., Chiozzi, P., Morelli, A., Passaro, A., Fellin, R. and Di Virgilio, F. (2003) Defective P2Y purinergic receptor function: A possible novel mechanism for impaired glucose transport. Journal of Cellular Physiology, 197, 435-444. doi:10.1002/jcp.10379
|
[13]
|
Misra, P. and Chakrabarti, R. (2007) The role of AMP kinase in diabetes. Indian Journal of Medical Research, 125, 389-398.
|
[14]
|
Miranda, N., Tovar, A.R., Palacios, B. and Torres, N. (2007) AMPK as a cellular energy sensor and its function in the organism. Revista de Investigacion Clinica, 59, 458- 469.
|
[15]
|
Remedi, M.S. and Koster, J.C. (2010) K(ATP) channelopathies in the pancreas. Pflugers Archiv, 460, 307-320.
|
[16]
|
Wasada, T. (2002) Adenosine triphosphate-sensitive potassium (K(ATP)) channel activity is coupled with insulin resistance in obesity and type 2 diabetes mellitus. Internal Medicine, 41, 84-90.
|
[17]
|
Chistiakov, D.A., Potapov, V.A., Khodirev, D.C., Shamkhalova, M.S., Shestakova, M.V. and Nosikov, V.V. (2009) Genetic variations in the pancreatic ATP-sensitive potassium channel, beta-cell dysfunction, and susceptibility to type 2 diabetes. Acta Diabetol, 46, 43-49.
|