|
[1]
|
Risk factors for cognitive impairment in middle-aged type 2 diabetic patients: a cross-sectional study
BMJ Open,
2024
DOI:10.1136/bmjopen-2023-074753
|
|
|
|
|
[2]
|
Peroxisom proliferator-activated receptor-γ coactivator-1α in neurodegenerative disorders: A promising therapeutic target
Biochemical Pharmacology,
2023
DOI:10.1016/j.bcp.2023.115717
|
|
|
|
|
[3]
|
Glucose metabolism in posterior cingulate cortex has supplementary value to predict the progression of cognitively unimpaired to dementia due to Alzheimer’s disease: an exploratory study of 18F-FDG-PET
GeroScience,
2023
DOI:10.1007/s11357-023-00897-0
|
|
|
|
|
[4]
|
Peroxisom proliferator-activated receptor-γ coactivator-1α in neurodegenerative disorders: A promising therapeutic target
Biochemical Pharmacology,
2023
DOI:10.1016/j.bcp.2023.115717
|
|
|
|
|
[5]
|
Glucose metabolism in posterior cingulate cortex has supplementary value to predict the progression of cognitively unimpaired to dementia due to Alzheimer’s disease: an exploratory study of 18F-FDG-PET
GeroScience,
2023
DOI:10.1007/s11357-023-00897-0
|
|
|
|
|
[6]
|
Potential for Ketotherapies as Amyloid-Regulating Treatment in Individuals at Risk for Alzheimer’s Disease
Frontiers in Neuroscience,
2022
DOI:10.3389/fnins.2022.899612
|
|
|
|
|
[7]
|
Phosphorus metabolism in the brain of cognitively normal midlife individuals at risk for Alzheimer's disease
Neuroimage: Reports,
2022
DOI:10.1016/j.ynirp.2022.100121
|
|
|
|
|
[8]
|
Mitochondrial Alterations in Neurons Derived from the Murine AppNL-F Knock-In Model of Alzheimer’s Disease
Journal of Alzheimer's Disease,
2022
DOI:10.3233/JAD-220383
|
|
|
|
|
[9]
|
Phosphorus metabolism in the brain of cognitively normal midlife individuals at risk for Alzheimer's disease
Neuroimage: Reports,
2022
DOI:10.1016/j.ynirp.2022.100121
|
|
|
|
|
[10]
|
Effects of non-modifiable risk factors of Alzheimer’s disease on intracortical myelin content
Alzheimer's Research & Therapy,
2022
DOI:10.1186/s13195-022-01152-y
|
|
|
|
|
[11]
|
Phosphorus metabolism in the brain of cognitively normal midlife individuals at risk for Alzheimer's disease
Neuroimage: Reports,
2022
DOI:10.1016/j.ynirp.2022.100121
|
|
|
|
|
[12]
|
Mitochondrial dysfunction, oxidative stress, neuroinflammation, and metabolic alterations in the progression of Alzheimer’s disease: A meta-analysis of in vivo magnetic resonance spectroscopy studies
Ageing Research Reviews,
2021
DOI:10.1016/j.arr.2021.101503
|
|
|
|
|
[13]
|
Mitochondrial dysfunction, oxidative stress, neuroinflammation, and metabolic alterations in the progression of Alzheimer’s disease: A meta-analysis of in vivo magnetic resonance spectroscopy studies
Ageing Research Reviews,
2021
DOI:10.1016/j.arr.2021.101503
|
|
|
|
|
[14]
|
Rosemary extract increases neuronal cell glucose uptake and activates AMPK
Applied Physiology, Nutrition, and Metabolism,
2021
DOI:10.1139/apnm-2020-0014
|
|
|
|
|
[15]
|
Uncovering Disease Mechanisms in a Novel Mouse Model Expressing Humanized APOEε4 and Trem2*R47H
Frontiers in Aging Neuroscience,
2021
DOI:10.3389/fnagi.2021.735524
|
|
|
|
|
[16]
|
Single-walled and multiwalled carbon nanotubes induce oxidative stress in isolated rat brain mitochondria
Toxicology and Industrial Health,
2019
DOI:10.1177/0748233719856983
|
|
|
|
|
[17]
|
Imagerie métabolique et moléculaire : l’exemple des maladies neurodégénératives
Bulletin de l'Académie Nationale de Médecine,
2018
DOI:10.1016/S0001-4079(19)30217-1
|
|
|
|
|
[18]
|
Oxidant/Antioxidant Imbalance in Alzheimer’s Disease: Therapeutic and Diagnostic Prospects
Oxidative Medicine and Cellular Longevity,
2018
DOI:10.1155/2018/6435861
|
|
|
|
|
[19]
|
Coupled Imaging with [18F]FBB and [18F]FDG in AD Subjects Show a Selective Association Between Amyloid Burden and Cortical Dysfunction in the Brain
Molecular Imaging and Biology,
2018
DOI:10.1007/s11307-018-1167-1
|
|
|
|
|
[20]
|
The spectrum of preclinical Alzheimer’s pathology and its modulation by ApoE genotype
Neurobiology of Aging,
2018
DOI:10.1016/j.neurobiolaging.2018.07.007
|
|
|
|
|
[21]
|
Synergistic interaction between APOE and family history of Alzheimer’s disease on cerebral amyloid deposition and glucose metabolism
Alzheimer's Research & Therapy,
2018
DOI:10.1186/s13195-018-0411-x
|
|
|
|
|
[22]
|
Individual cognitive and depressive traits associated with maternal versus paternal family history of Late-onset Alzheimer’s disease: Proactive semantic interference versus standard neuropsychological assessments
Personalized Medicine in Psychiatry,
2018
DOI:10.1016/j.pmip.2018.09.002
|
|
|
|
|
[23]
|
Atypical Localization and Dissociation between Glucose Uptake and Amyloid Deposition in Cognitively Normal APOE*E4 Homozygotic Elders Compared with Patients with Late-Onset Alzheimer’s Disease
eneuro,
2018
DOI:10.1523/ENEURO.0396-17.2018
|
|
|
|
|
[24]
|
Atypical Localization and Dissociation between Glucose Uptake and Amyloid Deposition in Cognitively Normal APOE*E4 Homozygotic Elders Compared with Patients with Late-Onset Alzheimer’s Disease
eneuro,
2018
DOI:10.1523/ENEURO.0396-17.2018
|
|
|
|
|
[25]
|
The starving brain: Overfed meets undernourished in the pathology of mild cognitive impairment (MCI) and Alzheimer's disease (AD)
Neurochemistry International,
2017
DOI:10.1016/j.neuint.2017.09.004
|
|
|
|
|
[26]
|
Pumping the Brakes: Neurotrophic Factors for the Prevention of Cognitive Impairment and Dementia after Traumatic Brain Injury
Journal of Neurotrauma,
2017
DOI:10.1089/neu.2016.4589
|
|
|
|
|
[27]
|
Molecular Imaging and Precision Medicine in Dementia and Movement Disorders
PET Clinics,
2017
DOI:10.1016/j.cpet.2016.08.003
|
|
|
|
|
[28]
|
Association between Cerebral Amyloid Deposition and Clinical Factors Including Cognitive Function in Geriatric Depression: Pilot Study Using Amyloid Positron Emission Tomography
Clinical Psychopharmacology and Neuroscience,
2016
DOI:10.9758/cpn.2016.14.4.378
|
|
|
|
|
[29]
|
Mitochondrial Dysfunction in Alzheimer’s Disease and the Rationale for Bioenergetics Based Therapies
Aging and disease,
2016
DOI:10.14336/AD.2015.1007
|
|
|
|
|
[30]
|
Cross-validation of biomarkers for the early differential diagnosis and prognosis of dementia in a clinical setting
European Journal of Nuclear Medicine and Molecular Imaging,
2016
DOI:10.1007/s00259-015-3170-y
|
|
|
|
|
[31]
|
Advances in PET Imaging of Degenerative, Cerebrovascular, and Traumatic Causes of Dementia
Seminars in Nuclear Medicine,
2016
DOI:10.1053/j.semnuclmed.2015.09.003
|
|
|
|
|
[32]
|
Mitochondrial Dysfunction in Neurodegenerative Disorders
2016
DOI:10.1007/978-3-319-28637-2_5
|
|
|
|
|
[33]
|
Impact of spillover from white matter by partial volume effect on quantification of amyloid deposition with [11C]PiB PET
NeuroImage,
2016
DOI:10.1016/j.neuroimage.2016.09.028
|
|
|
|
|
[34]
|
The Use of Neuroimaging to Assess Associations Among Diet, Nutrients, Metabolic Syndrome, and Alzheimer’s Disease
Journal of Alzheimer's Disease,
2015
DOI:10.3233/JAD-150301
|
|
|
|
|
[35]
|
APOε2 and education in cognitively normal older subjects with high levels of AD pathology at autopsy: findings from the Nun Study
Oncotarget,
2015
DOI:10.18632/oncotarget.4118
|
|
|
|