Journal of Cancer Therapy

Journal of Cancer Therapy

ISSN Print: 2151-1934
ISSN Online: 2151-1942
www.scirp.org/journal/jct
E-mail: jct@scirp.org
"Normal Human Cell Conversion to 3-D Cancer-like Growth: Genome Damage, Endopolyploidy, Senecence Escape, and Cell Polarity Change/Loss"
written by Kirsten H. Walen,
published by Journal of Cancer Therapy, Vol.2 No.2, 2011
has been cited by the following article(s):
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[1] CELL CYCLE STRESS IN NORMAL HUMAN CELLS: A ROUTE TO “FIRST CELLS”(WITH/WITHOUT FITNESS GAIN) AND CANCER-LIKE CELL-SHAPE CHANGES
2021
[2] A Traceable Cancer Model: DNA Damage, Fragile Site-SMGs, Mitotic Slippage, 4n-Genome-Reduction to Fitness-Gained, Initiating, 2n First Cells
2021
[3] Near-Dead Cells to Special Tetraploidy to First Cells to Cancer Diagnostic Morphology: Unlikely Therapy-Gain from For-Profit Industrial Goliath
2020
[4] Genomic Instability in Cancer II: 4N-Skewed (90°) Reductive Division via Fragile Sites to Fitness Increase for Solid and Hematological Cancer Beginnings
2019
[5] Cancer cells arise from bacteria
2018
[6] Genomic Instability in Cancer I: DNA-Repair Triggering Primitive Hereditary 4n-Skewed, Amitotic Division-System, the Culprit in EMT/MET/Metaplasia Cancer …
2018
[7] Mitotic Slippage Process Concealed Cancer-Sought Chromosome Instability Mechanism (S-CIN)
2017
[8] Cancer Prevention? Fundamental Genomic Alterations Are Present in Preneoplasia, Including Function of High Frequency Selected Mutations (HFSMs)
2016
[9] Wound Healing Is a First Response in a Cancerous Pathway: Hyperplasia Developments to 4n Cell Cycling in Dysplasia Linked to Rb-Inactivation
Journal of Cancer Therapy, 2015
[10] Neoplastic-Like CELL Changes of Normal Fibroblast Cells Associated with Evolutionary Conserved Maternal and Paternal Genomic Autonomous Behavior (Gonomery)
Journal of Cancer Therapy, 2014
[11] Neoplastic-Like Cell Changes of Normal Fibroblast Cells Associated with Evolutionary Conserved Maternal and Paternal Genomic Autonomous Behavior …
2014
[12] Haploidization of Human Diploid Metaphase Cells: Is This Genome Reductive Mechanism Opperational in Near-Haploid Leukemia
2014
[13] Normal Human Cells Acquiring Proliferative Advantage to Hyperplasia-Like Growth-Morphology: Aberrant Progeny Cells Associated with Endopolyploid …
2013
[14] Normal human cells acquiring proliferative advantage to hyperplasia-like growth-morphology: Aberrant progeny cells associated with endopolyploid and …
2013
[15] Normal Human Cells Acquiring Proliferative Advantage to Hyperplasia-Like Growth-Morphology: Aberrant Progeny Cells Associated with Endopolyploid and Haploid …
Cancer and Clinical Oncology, 2013
[16] Senescence Arrest of Endopolyploid Cells Renders Senescence into One Mechanism for Positive Tumorigenesis
Tumor Dormancy, Quiescence, and Senescence, Volume 1. Springer Netherlands,?, 2013
[17] into One Mechanism for Positive Tumorigenesis
Tumor Dormancy, Quiescence, and Senescence, Volume 1: Aging, Cancer, and Noncancer Pathologies?, 2013
[18] Normal Human Cells Acquiring Proliferative Advantage to Hyperplasia-Like Growth-Morphology: Aberrant Progeny Cells Associated with Endopolyploid and Haploid Divisions
Cancer and Clinical Oncology, 2013
[19] Haploidization of Human Diploid Metaphase Cells: Is This Genome Reductive Mechanism Opperational in Near-Haploid Leukemia?
Journal of Cancer Therapy, 2013
[20] Genome reversion process of endopolyploidy confers chromosome instability on the descendent diploid cells
Cell biology international?, 2012
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