"The chemical mechanism of oxidative stress due to the non-transferrin-bound iron (NTBI)"
written by Yuzo Nishida,
published by Advances in Bioscience and Biotechnology, Vol.3 No.7A, 2012
has been cited by the following article(s):
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[2] Iron-mediated oxidative cell death is a potential contributor to neuronal dysfunction induced by neonatal hemolytic hyperbilirubinemia
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[3] Current insights on the role of iron and copper dyshomeostasis in the pathogenesis of bilirubin neurotoxicity.
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[4] Current insights on the role of iron and copper dyshomeostasis in the pathogenesis of bilirubin neurotoxicity
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[5] 鉄代謝異常の解明に向けて~ 新たな創薬のターゲット~
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[6] 鉄化合物の構造予測と発ガン性
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[7] Abnormal iron accumulation in brain and new iron chelator for labile iron removal therapy
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[8] IRON CHELATORS FOR LABILE IRON REMOVAL THERAPY AND ENHANCEMENT OF LONGEVITY
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[9] Investigated and available therapeutic options for treating aceruloplasminemia
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[10] PROTECTIVE EFFECTS OF HYDROGEN MOLECULE AND LABILE IRON REMOVAL THERAPY AGAINST NEPHROTOXICITY BY CIS-PLATIN
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[11] New Insight into Toxicity due to Oligomeric Amyloid-[Beta] Peptide and Alpha-Synuclein Protein induced by Copper (II) ion
International Journal of Chemistry, 2015
[12] Abnormal Iron Accumulation in Brain and New Iron Chelator for Labile Iron Removal Theraphy
International Journal of Chemistry, 2015
[13] New Insight into Toxicity due to Oligomeric Amyloid-b Peptide and Alpha-Synuclein Protein induced by Copper (II) ion
International Journal of Chemistry, 2014
[14] Model Reactions for the Formation of Iron Deposition
International Journal of Chemistry, 2014