"Increased survivin expression contributes to apoptosis-resistance in IPF fibroblasts"
written by Thomas H. Sisson, Toby M. Maher, Iyabode O. Ajayi, Jessie E. King, Peter D.R. Higgins, Adam J. Booth, Rommel L. Sagana, Steven K. Huang, Eric S. White, Bethany B. Moore, Jeffrey C. Horowitz,
published by Advances in Bioscience and Biotechnology, Vol.3 No.6A, 2012
has been cited by the following article(s):
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[10] FoxM1-dependent RAD51 and BRCA2 signaling protects idiopathic pulmonary fibrosis fibroblasts from radiation-induced cell death
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[12] Glutaminolysis Epigenetically Regulates Anti-Apoptotic Gene Expression in IPF Fibroblasts
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[13] Survivin IPF: Targeting Cellular Metabolism to Promote Apoptosis in IPF Fibroblasts
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[17] Mouse Lung Fibroblast Resistance to Fas-Mediated Apoptosis Is Dependent on the Baculoviral Inhibitor of Apoptosis Protein 4 and the Cellular FLICE …
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[18] TGF-β1 Signaling and Tissue Fibrosis
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[19] Mouse Lung Fibroblast Resistance to Fas-Mediated Apoptosis Is Dependent on the Baculoviral Inhibitor of Apoptosis Protein 4 and the Cellular FLICE- …
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[20] Survivin in autoimmune diseases
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[21] Lung carcinogenesis and fibrosis taken together: just coincidence?
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[22] Lung carcinogenesis and fibrosis taken together: just co-incidence?
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[23] Wenyang Huazhuo Tongluo formula, a Chinese herbal decoction, improves skin fibrosis by promoting apoptosis and inhibiting proliferation through down …
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[25] Survivin 与肺部疾病关系的研究进展
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[26] MicroRNA-29c regulates apoptosis sensitivity via modulation of the cell-surface death receptor, Fas, in lung fibroblasts
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[27] Fibrocytes, γ-Herpesvirus and Periostin Augment Bleomycin-induced Lung Fibrosis
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[30] Wenyang Huazhuo Tongluo formula, a Chinese herbal decoction, improves skin fibrosis by promoting apoptosis and inhibiting proliferation through down-regulation of survivin and cyclin D1 in systemic sclerosis
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[36] Macitentan reverses early obstructive pulmonary vasculopathy in rats: early intervention in overcoming the survivin-mediated resistance to apoptosis
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[38] Myofibroblast Dedifferentiation by PGE2 is Characterized by Reversal of the Global Transcriptome
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[39] Aberrant expression and activity of histone deacetylases in sporadic idiopathic pulmonary fibrosis
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[40] Inhibition of Cyclic Adenosine Monophosphate (cAMP)-Response Element-Binding Protein (CREB)-Binding Protein (CBP)/β-Catenin Reduces Liver Fibrosis in Mice
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[41] Plakoglobin expression in fibroblasts and its role in idiopathic pulmonary fibrosis
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[42] Fibroblast biology in idiopathic pulmonary fibrosis
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[43] 慢病毒介导生存素基因转染反分化早期和晚期椎间盘髓核细胞生物学效应的比较
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[44] Targeting Inhibitor Of Apoptosis Proteins Protects From Bleomycin-Induced Lung Fibrosis
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[45] Inhibition of cyclic adenosine monophosphate (cAMP)-response element-binding protein (CREB)-binding protein (CBP)/β-catenin reduces liver fibrosis in …
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[46] Lung Fibroblasts from Patients with Idiopathic Pulmonary Fibrosis Exhibit
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[47] Alveolar epithelial cells in idiopathic pulmonary fibrosis display upregulation of TRAIL, DR4 and DR5 expression with simultaneous preferential over …
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[48] Lung fibroblasts from patients with idiopathic pulmonary fibrosis exhibit genome-wide differences in DNA methylation compared to fibroblasts from nonfibrotic …
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[49] The epidemiological situation of tuberculosis in Poland: Part I. According to notification rates, the incidence of tuberculosis varies in different regions of …
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[50] Immunopathogenesis of idiopathic pulmonary fibrosis
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[51] Immunopatogeneza samoistnego włóknienia płuc
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[52] Mesenchymal stem cell-like properties in fibroblasts
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[53] Lung fibroblasts from patients with idiopathic pulmonary fibrosis exhibit genome-wide differences in DNA methylation compared to fibroblasts from nonfibrotic …
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[54] Alveolar epithelial cells in idiopathic pulmonary fibrosis display upregulation of TRAIL, DR4 and DR5 expression with simultaneous preferential over-expression …
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[55] Pathogenesis of idiopathic pulmonary fibrosis
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[56] Following the Path of CCL2 from Prostaglandins to Periostin in Lung Fibrosis
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[57] Expression of the apoptosis inhibitor livin in colorectal adenoma-carcinoma sequence: correlations with pathology and outcome
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[58] Dual Targeting of MEK and PI3K Pathways Attenuates Established and Progressive Pulmonary Fibrosis
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[59] Clinical aspects for survivin: a crucial molecule for targeting drug-resistant cancers
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[60] Mesenchymal Stem Cell-Like Properties in Fibroblasts.
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[61] Lung Fibroblasts from Patients with Idiopathic Pulmonary Fibrosis Exhibit Genome-Wide Differences in DNA Methylation Compared to Fibroblasts from Nonfibrotic Lung
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[62] Alveolar epithelial cells in idiopathic pulmonary fibrosis display upregulation of TRAIL, DR4 and DR5 expression with simultaneous preferential over-expression of pro-apoptotic marker p53
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[63] Outstaying their Welcome: The Persistent Myofibroblast in IPF
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[68] Arsenic trioxide induces apoptosis in B-cell chronic lymphocytic leukemic cells through down-regulation of survivin via the p53-dependent signaling pathway
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[69] X-linked inhibitor of apoptosis regulates lung fibroblast resistance to Fas-mediated apoptosis
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[70] Histone modifications are responsible for decreased Fas expression and apoptosis resistance in fibrotic lung fibroblasts
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[72] Novel combination of collagen dynamics analysis and transcriptional profiling reveals fibrosis-relevant genes and pathways
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[73] Cell matrix interactions in lung fibrosis
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[74] CERTIFICATIONS AND LICENSURES
2005
[75] Jeffrey C. Horowitz, MD, FCCP
Ann Arbor, 2005
[76] Certification and Licensure
Ann Arbor, 2005