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Helicobacter pylori LPS-induced gastric mucosal spleen tyrosine kinase (Syk) recruitment to TLR4 and activation occurs with the involvement of protein kinase Cδ
Inflammopharmacology,
2018
DOI:10.1007/s10787-017-0430-4
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[2]
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Mitochondrial Membranes Restitution Proceeds via Vesicular Import from ER and Cytosol. Counterparts’ Resemblances and Variances in Mitochondria and Golgi Pathways
Advances in Biological Chemistry,
2017
DOI:10.4236/abc.2017.71001
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[3]
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Tissue repair in myxobacteria: A cooperative strategy to heal cellular damage
BioEssays,
2016
DOI:10.1002/bies.201500132
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[4]
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Helicobacter pylori-induced gastric mucosal TGF-α ectodomain shedding and EGFR transactivation involves Rac1/p38 MAPK-dependent TACE activation
Inflammopharmacology,
2016
DOI:10.1007/s10787-015-0254-z
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[5]
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Tissue repair in myxobacteria: A cooperative strategy to heal cellular damage
BioEssays,
2016
DOI:10.1002/bies.201500132
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[6]
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Helicobacter pylori-elicited induction in gastric mucosal matrix metalloproteinase-9 (MMP-9) release involves ERK-dependent cPLA2 activation and its recruitment to the membrane-localized Rac1/p38 complex
Inflammopharmacology,
2016
DOI:10.1007/s10787-016-0261-8
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Role of amplification in phospholipase Cγ2 activation in modulation of gastric mucosal inflammatory responses to Helicobacter pylori: effect of ghrelin
Inflammopharmacology,
2015
DOI:10.1007/s10787-014-0220-1
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[8]
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Mechanism of Rac1-induced amplification in gastric mucosal phospholipase Cγ2 activation in response to Helicobacter pylori: modulatory effect of ghrelin
Inflammopharmacology,
2015
DOI:10.1007/s10787-015-0231-6
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[9]
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Porphyromonas gingivalis-Induced GEF Dock180 Activation by Src/PKCδ-Dependent Phosphorylation Mediates PLCγ2 Amplification in Salivary Gland Acinar Cells: Modulatory Effect of Ghrelin
Journal of Biosciences and Medicines,
2015
DOI:10.4236/jbm.2015.37008
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[10]
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Porphyromonas gingivalis-Stimulated TACE Activation for TGF-α Ectodomain Shedding and EGFR Transactivation in Salivary Gland Cells Requires Rac1-Dependent p38 MAPK Membrane Localization
Journal of Biosciences and Medicines,
2015
DOI:10.4236/jbm.2015.311005
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[11]
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Protein Kinase Cδ-Mediated Posttranslational Phosphorylation of Constitutive Nitric Oxide Synthase Regulates Gastric Mucosal Inflammatory Responses to Helicobacter pylori: Effect of Ghrelin
Journal of Biosciences and Medicines,
2014
DOI:10.4236/jbm.2014.25003
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[12]
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Modulation of gastric mucosal inflammatory responses to Helicobacter pylori via ghrelin-induced protein kinase Cδ tyrosine phosphorylation
Inflammopharmacology,
2014
DOI:10.1007/s10787-014-0206-z
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Role of ghrelin-induced phosphatidylinositol 3-kinase activation in modulation of gastric mucosal inflammatory responses to Helicobacter pylori
Inflammopharmacology,
2014
DOI:10.1007/s10787-013-0190-8
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[14]
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Concluding Step in Cell Restitution Cycle: ER Transport Vesicles with Sphingolipids in the Outer Leaflet of the Membrane Restore Lysosomes
Advances in Biological Chemistry,
2014
DOI:10.4236/abc.2014.45036
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Homeostatic cell cycle and the origin of autophagosomal vesicles
Advances in Biological Chemistry,
2013
DOI:10.4236/abc.2013.33031
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